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R manipulating IGFBP3 levels in two GIST cell lines, we observed a modulating effect on response in GIST882, suggesting that the induction of IGFBP3 is a significant, specific response to imatinib-induced stress. Failure to observe a similar response in GIST-T1 suggested that GIST-T1 cells are insensitive to IGFBP3. However, additional studies showed that IGFBP3 regulates GIST cell viability with

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